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Stéphanie Larose Division of Endocrinology, Department of Medicine, Centre hospitalier de l’Université de Montréal (CHUM), Université de Montréal, Montréal, QC, Canada

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Dany Rioux Division of Endocrinology, Department of Medicine, Centre hospitalier universitaire régional, Trois-Rivières, QC, Canada

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Roula Albadine Department of Pathology, Centre hospitalier de l’Université de Montréal, Université de Montréal (CHUM), Université de Montréal, Montréal, QC, Canada, Montréal, QC, Canada

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André Lacroix Division of Endocrinology, Department of Medicine, Centre hospitalier de l’Université de Montréal (CHUM), Université de Montréal, Montréal, QC, Canada

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recurrence can occur after a long delay. Background Ectopic adrenocorticotrophic hormone (ACTH) secretion (EAS) is a rare cause of ACTH-dependent Cushing’s syndrome (CS), most often caused by a thoracic neuroendocrine tumor (NET) ( Frete et al

Article Type:
Case Report
DOI:
https://doi.org/10.1530/EO-23-0002
Volume/Issue:
Volume 3: Issue 1
Open access
Laura Gerard Départment d’Oncologie Médicale, Hôpital Edouard Herriot, Hospices Civils de Lyon, Lyon, France
European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France

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David Barthelemy CIRCAN (CIRculating CANcer) Platform, Hospices Civils de Lyon, GHS University Hospital, Pierre-Benite, France

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Arnaud Gauthier CIRCAN (CIRculating CANcer) Platform, Hospices Civils de Lyon, GHS University Hospital, Pierre-Benite, France

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Valerie Hervieu European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France
Institut Multi-Site de Pathologie et Biologie Moléculaire, Groupement Hospitalier Est, Hospices Civils de Lyon, Bron, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France

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Jonathan Lopez European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France
Institut Multi-Site de Pathologie et Biologie Moléculaire, Groupement Hospitalier Est, Hospices Civils de Lyon, Bron, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France

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Benjamin Gibert INSERM, UMR 1052, Lyon Cancer Research Center, Faculté Laennec, Lyon Cedex 08, France

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Helene Lasolle European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France
Fédération d’Endocrinologie, Groupement Hospitalier Est, Hospices Civils de Lyon, Bron Cedex, France

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Laurence Chardon Fédération de Biochimie, Groupement Hospitalier Est, Hospices Civils de Lyon, Bron Cedex, France

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Jessica Garcia CIRCAN (CIRculating CANcer) Platform, Hospices Civils de Lyon, GHS University Hospital, Pierre-Benite, France

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Gérald Raverot European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France
INSERM, UMR 1052, Lyon Cancer Research Center, Faculté Laennec, Lyon Cedex 08, France
Fédération d’Endocrinologie, Groupement Hospitalier Est, Hospices Civils de Lyon, Bron Cedex, France

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Léa Payen CIRCAN (CIRculating CANcer) Platform, Hospices Civils de Lyon, GHS University Hospital, Pierre-Benite, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France

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Thomas Walter Départment d’Oncologie Médicale, Hôpital Edouard Herriot, Hospices Civils de Lyon, Lyon, France
European NeuroEndocrine Tumours Society (ENETS) Lyon Center of Excellence, Lyon, France
Université de Lyon, Université Claude Bernard Lyon 1, Villeurbanne Cedex, France
INSERM, UMR 1052, Lyon Cancer Research Center, Faculté Laennec, Lyon Cedex 08, France

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consistent with Cushing’s syndrome, which was confirmed: serum cortisol level was 57.7 µg/dL (normal level 10–20 µg/dL), 24-h urine free cortisol (UFC) measurement was 27,306 µg/24 h (normal level 4.3–176 µg/24 h), and adrenocorticotropic hormone (ACTH) level

Article Type:
Case Report
DOI:
https://doi.org/10.1530/EO-21-0012
Volume/Issue:
Volume 1: Issue 1
Open access
D Mc Donald Academic Department of Endocrinology, Beaumont Hospital, Dublin, Ireland

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M Sherlock Academic Department of Endocrinology, Beaumont Hospital, Dublin, Ireland

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C J Thompson Academic Department of Endocrinology, Beaumont Hospital, Dublin, Ireland

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survival in cancer improves, the negative effects of hyponatraemia on quality of life are also assumed importance. The role of ectopic secretion of the antidiuretic hormone, arginine vasopressin (AVP), in causing hyponatraemia due to the syndrome of

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EO-22-0056
Volume/Issue:
Volume 2: Issue 1
Open access
Katherine A English OCDEM, Radcliffe Department of Medicine, University of Oxford, Churchill Hospital, Oxford, UK

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Rajesh V Thakker OCDEM, Radcliffe Department of Medicine, University of Oxford, Churchill Hospital, Oxford, UK
Oxford NIHR Biomedical Research Centre, Oxford University Hospitals Trust, Oxford, UK

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Kate E Lines OCDEM, Radcliffe Department of Medicine, University of Oxford, Churchill Hospital, Oxford, UK
Oxford NIHR Biomedical Research Centre, Oxford University Hospitals Trust, Oxford, UK

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secretion ( Hackeng et al. 2020 ). Another example of an inappropriate gain in secretory properties of PNETs is ectopic adrenocorticotrophin (ACTH)-secreting PNETs. One study reported lower pro-opiomelanocortin methylation of seven ACTH-PNETs when compared

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EO-23-0003
Volume/Issue:
Volume 3: Issue 1
Open access