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Erika Peverelli Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy

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Donatella Treppiedi Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy

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Giovanna Mantovani Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy
Fondazione IRCCS Ca’ Granda Ospedale Maggiore Policlinico, Endocrinology Unit, Milan, Italy

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ubiquitin-specific peptidase 8 (USP8) have been shown to regulate SSTR5 in corticotroph tumors, suggesting clinical implications in the pharmacological responsiveness to pasireotide treatment. Role of cytoskeleton protein FLNA FLNA is a large

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EO-22-0042
Volume/Issue:
Volume 2: Issue 1
Open access
Sylvia L Asa Department of Pathology, University Hospitals Cleveland Medical Center, Case Western Reserve University, Cleveland, Ohio, USA

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Shereen Ezzat Department of Medicine, University Health Network, University of Toronto, Toronto, Ontario, Canada

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activating mutations of GNAS that result in constitutive activation of cyclic AMP signaling and some corticotroph tumors have mutations of the USP8 or USP48 genes ( Ezzat et al. 2018 , Asa et al. 2021 c ); occasional aggressive corticotroph

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EO-22-0041
Volume/Issue:
Volume 2: Issue 1
Open access
Sandra Pekic School of Medicine, University of Belgrade, Belgrade, Serbia
Clinic for Endocrinology, Diabetes and Metabolic Diseases, University Clinical Center Belgrade, Belgrade, Serbia

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Marko Stojanovic School of Medicine, University of Belgrade, Belgrade, Serbia
Clinic for Endocrinology, Diabetes and Metabolic Diseases, University Clinical Center Belgrade, Belgrade, Serbia

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Vera Popovic School of Medicine, University of Belgrade, Belgrade, Serbia

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identified in 30% of somatotropinomas, GPR101 mutation identified in 4% of somatotropinomas and USP8 mutation identified in 40% of corticotrophinomas, are not clearly associated with tumor behavior ( Reincke et al. 2015 , Chen et al. 2018 , Sbiera

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EO-21-0033
Volume/Issue:
Volume 2: Issue 1
Open access
Eleanor Fewings Academic Department of Medical Genetics, National Institute for Health Research Cambridge Biomedical Research Centre, University of Cambridge, Cambridge, UK

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Serena Khoo Sert Kim Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Alexey Larionov Academic Department of Medical Genetics, National Institute for Health Research Cambridge Biomedical Research Centre, University of Cambridge, Cambridge, UK

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Alison Marker Department of Histopathology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Olivier Giger Department of Pathology, University of Cambridge, Cambridge, UK

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Ashley Shaw Department of Radiology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Graeme R Clark Academic Department of Medical Genetics, National Institute for Health Research Cambridge Biomedical Research Centre, University of Cambridge, Cambridge, UK

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Vasilis Kosmoliaptsis Department of Surgery, University of Cambridge and NIHR Cambridge Biomedical Research Centre, Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK

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Benjamin G Challis Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Marc Tischkowitz Academic Department of Medical Genetics, National Institute for Health Research Cambridge Biomedical Research Centre, University of Cambridge, Cambridge, UK
East Anglian Medical Genetics Service, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Ruth T Casey Academic Department of Medical Genetics, National Institute for Health Research Cambridge Biomedical Research Centre, University of Cambridge, Cambridge, UK
Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Case 6 Case 7 Case 8 Case 9 a Case 10 LWB criteria Malignant Malignant Malignant Malignant Malignant Malignant Malignant Malignant Borderline Benign Age (years) 66 43 55 71 58 72 71 46 59

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EO-21-0011
Volume/Issue:
Volume 1: Issue 1
Open access